"jamie" wrote

In persons capable of producing insulin (not Type I diabetics), there
are feedback mechanisms to prevent blood ketone levels going too high
and causing ketoacidosis. The body slows or stops metabolizing fats,
and releases insulin to use glucose (if not from carb, then from
gluconeogenesis breakdown of proteins).

Thanks, Jamie. I think in the context of the thread what Doug may also have
been getting at was the role of Acylation stimulation protein (ASP). DJ
provided a link to:

http://www.bodyrecomposition.com/for...ead.php?t=2689

which I found pretty interesting. I guess either or both could apply,
depending on the usual other variables.

HG