Well DUH! Almonds contain fiber people. Fats bond to fiber and they all
don't get digested, hence while you might be consuming through the mouth the
same amount of calories, your body isn't.

Again, very misleading article.

Trent

But if what you say is true, then the article is correct. People who
are eating almonds and the like -- by your reasoning -- are taking in
more calories but digesting less of them. But then you'd have to
deal with the fact that the "low fat" diet folks are also eating fiber
(I assume), so the fiber must also be removing their low amounts of
fat. What's the difference?

LOL You might be interested in this study on the interaction between
obesity genes and dietary fat. Perhaps this is one reason why there is
so much disagreement about weight loss methods. There are hundreds of
obesity-causing genetic defects and, like poker players, we are all
dealt a different genetic hand, which accounts for the large
variations in the way people respond to diet, exercise, weight loss
drugs, etc. But people tend to think that what works for them will
work for everybody, so we end up with confusion and strife.

Nieters A, et al. "Polymorphisms in candidate obesity genes and their
interaction with dietary intake of n-6 polyunsaturated fatty acids
affect obesity risk in a sub-sample of the EPIC-Heidelberg cohort."
Eur J Nutr. 2002 Oct;41(5):210-21; PMID: 12395215.
Full abstract: http://tinyurl.com/w9or
"BACKGROUND, AIM: In several genes coding for molecules involved in
the regulation of body weight (fat mass) and thermogenesis,
polymorphisms have been reported which possibly modify human obesity
risk . . . Importantly, the results of the analysis of gene-diet
interactions suggest that the allelic variants of candidate genes
(leptin, TNFA, PPARG2) might strongly affect diet-related obesity
risk. CONCLUSIONS: The results support some but not all previous
reports about a risk-modulating effect of polymorphisms in genes
affecting obesity risk. THE MOST IMPORTANT FINDING IS AN INDICATION OF
SUBSTANTIAL INTERACTION BETWEEN ALLELIC VARIANTS OF PARTICULAR GENES
AND FATTY ACID INTAKE-RELATED OBESITY RISK. These observations suggest
that future studies on polymorphisms in obesity genes should take data
on dietary habits into account." [emphasis added]

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