FOB (removethis) wrote:

Those are symptoms, not the item detected by the strips.

Eating excess protein does not lead to ketones that I have ever read.
Through glycation proteins are converted to glucose and glucose turns
off the production of ketones.

"When glycogen stores are not available in the cells, fat (triacylglycerol)
is cleaved to give 3 fatty acid chains and 1 glycerol molecule in a process
called lipolysis. Most of the body is able to utilize fatty acids as an
alternative source of energy in a process called beta-oxidation. One of the
products of beta-oxidation is acetyl-CoA, which can be further used in the
Krebs cycle. During prolonged fasting or starvation, acetyl-CoA in the liver
is used to produce ketone bodies instead, leading to a state of ketosis.
During starvation or a long physical training session, the body starts
utilizing fatty acids instead of glucose. The brain cannot use fatty acids
for energy because the fatty acids cannot cross the blood-brain barrier.
However, the ketone bodies produced in the liver can cross the blood-brain
barrier. In the brain, these ketone bodies are then incorporated into
acetyl-CoA and used in the Krebs cycle."

Level one, cutting the fat into the fatty acids and glycerol - Two
glycerols are combined to make a glucose. About 10% of the energy from
fat is in the form of this glucose. I tend to think of it as
establishing a background for glucose that's well above zero.

Level two, burning the fatty acids for fuel - You described the two
paths. The acetyl-CoA path does not generate any ketones that end up in
the urine and that's how fat can be burned on non-ketotic diet plans.
There's a speed limit to it. Marathon training works to increase that
speed limit. The ketone path is used as an additional method and so it
means when there are ketones in the urine your body is burning fat
faster than the limit of the acetyl-CoA method. It means faster.