"Many with type-2 diabetes have enough beta cells remaining to produce
enough endogenous insulin to maintain euglycemia once the VAT is gone
and the body regains normal insulin sensitivity."


You are woefully out of touch with the current literature on vat and
diabetes in general. Vat is a secondary effect of insulin resistance
not the cause of it. When developed it contributes to feed back to
resistance but is not the cause of it. As such it is not the cause of
diabetes and even when reduced diabetes remains. Such a black and white
single cause view of any part of the complexity of biology is a problem.