Death of a 16 -year-old girl on a low-carb diet.

In article . com "Andrew B. Chung, MD/PhD" writes:
convicted neighbor Cary Kittrell wrote:
Andrew, in the Holy Spirit, boldly wrote:
convicted neighbor TC wrote:
Andrew, in the Holy Spirit, boldly wrote:
friend Jim Chinnis wrote:
convicted neighbor Susan wrote:

You'd die without
fat or protein but live a very long life without dietary carbs.

True.

Was not true for this 16-year-old girl who forsaked dietary carbs (my
condolences to surviving friends and family):

"Emergency teams were summoned to a local high school to care for a 16-
year-old girl who had suddenly collapsed. She was 5 feet 8 inches tall
and weighed approximately 80 to 85 kg (176 to 187 lb). The patient had
been in good health with no known history of medical problems. She had
started a low-carbohydrate/high-protein diet 1 or 2 weeks earlier in
an effort to lose weight. She had learned about the diet from video
tapes, purchased from an advertisement on television. Her mother had
been on the same diet. She had complied with the dietary regimen,
eating meat, cheese, and salads without fasting. When the paramedics
arrived, the girl was pulseless and apneic. The electrocardiogram
revealed ventricular fibrillation. The patient's trachea was intubated
and cardiopulmonary resuscitation was initiated. Resuscitative
measures, including defibrillation, were without effect. On arrival in
the emergency department, the patient remained pulseless, with no
evidence of cardiac activity. Arterial blood gas analysis revealed a
pH of 6.89, with a base deficit of -19.8. Other laboratory values were
serum sodium 142 mEq/L, ionized calcium 1.12 mEq/L, and serum
potassium 3.8 mEq/L. Further resuscitative efforts were without
effect. Postmortem and toxicologic examinations revealed no apparent
cause for the death. Subsequent cardiologic evaluation of the
patient's 12-year-old sibling including echocardiography and
electrocardiography was within normal limits."

Source:

http://tinyurl.com/28ycon

Be hungry... be healthy... be blessed:

http://TheWellnessFoundation.com/PressRelease

Chung, you friggin' heartless moron. You are such a friggin'
opportunist.

Name-calling simply shows that you are lost:

http://HeartMDPhD.com/HolySpirit/dream.asp

Not only that, he misspelled "ghoul".

No.

In your case of cognitive deficits,

"In your case of cognitive defects"?

In some language that's English?

-- cary

(hi, Joss Whedon)

convicted neighbor Cary Kittrell wrote:
Andrew, in the Holy Spirit, boldly wrote:
convicted neighbor Cary Kittrell wrote:
Andrew, in the Holy Spirit, boldly wrote:
convicted neighbor TC wrote:
Andrew, in the Holy Spirit, boldly wrote:
friend Jim Chinnis wrote:
convicted neighbor Susan wrote:

You'd die without
fat or protein but live a very long life without dietary carbs.

True.

Was not true for this 16-year-old girl who forsaked dietary carbs (my
condolences to surviving friends and family):

"Emergency teams were summoned to a local high school to care for a 16-
year-old girl who had suddenly collapsed. She was 5 feet 8 inches tall
and weighed approximately 80 to 85 kg (176 to 187 lb). The patient had
been in good health with no known history of medical problems. She had
started a low-carbohydrate/high-protein diet 1 or 2 weeks earlier in
an effort to lose weight. She had learned about the diet from video
tapes, purchased from an advertisement on television. Her mother had
been on the same diet. She had complied with the dietary regimen,
eating meat, cheese, and salads without fasting. When the paramedics
arrived, the girl was pulseless and apneic. The electrocardiogram
revealed ventricular fibrillation. The patient's trachea was intubated
and cardiopulmonary resuscitation was initiated. Resuscitative
measures, including defibrillation, were without effect. On arrival in
the emergency department, the patient remained pulseless, with no
evidence of cardiac activity. Arterial blood gas analysis revealed a
pH of 6.89, with a base deficit of -19.8. Other laboratory values were
serum sodium 142 mEq/L, ionized calcium 1.12 mEq/L, and serum
potassium 3.8 mEq/L. Further resuscitative efforts were without
effect. Postmortem and toxicologic examinations revealed no apparent
cause for the death. Subsequent cardiologic evaluation of the
patient's 12-year-old sibling including echocardiography and
electrocardiography was within normal limits."

Source:

http://tinyurl.com/28ycon

Be hungry... be healthy... be blessed:

http://TheWellnessFoundation.com/PressRelease

Chung, you friggin' heartless moron. You are such a friggin'
opportunist.

Name-calling simply shows that you are lost:

http://HeartMDPhD.com/HolySpirit/dream.asp

Not only that, he misspelled "ghoul".

No.

In your case of cognitive deficits,

"In your case of cognitive defects"?

In some language that's English?

Yes, that would be the written language, dear Cary.

Many thanks, much praise, and all the glory to GOD for HIS compelling
you to unwittingly demonstrate how you have both cognitive deficits
and defects.

Be hungry... be healthy... be happy... be blessed:

Prayerfully in Jesus' awesome love,

Andrew
--
Andrew B. Chung, MD/PhD
Cardiologist

neighbor Michael Sand wrote:
Hi Andrew!

Andrew, in the Holy Spirit, boldly wrote:
friend Jim Chinnis wrote:
convicted neighbor Susan wrote:

You'd die without
fat or protein but live a very long life without dietary carbs.

True.

Was not true for this 16-year-old girl who forsaked dietary carbs (my
condolences to surviving friends and family):

"Emergency teams were summoned to a local high school to care for a 16-
year-old girl who had suddenly collapsed. She was 5 feet 8 inches tall
and weighed approximately 80 to 85 kg (176 to 187 lb). The patient had
been in good health with no known history of medical problems. She had
started a low-carbohydrate/high-protein diet 1 or 2 weeks earlier in
an effort to lose weight. She had learned about the diet from video
tapes, purchased from an advertisement on television. Her mother had
been on the same diet. She had complied with the dietary regimen,
eating meat, cheese, and salads without fasting. When the paramedics
arrived, the girl was pulseless and apneic. The electrocardiogram
revealed ventricular fibrillation. The patient's trachea was intubated
and cardiopulmonary resuscitation was initiated. Resuscitative
measures, including defibrillation, were without effect. On arrival in
the emergency department, the patient remained pulseless, with no
evidence of cardiac activity. Arterial blood gas analysis revealed a
pH of 6.89, with a base deficit of -19.8. Other laboratory values were
serum sodium 142 mEq/L, ionized calcium 1.12 mEq/L, and serum
potassium 3.8 mEq/L. Further resuscitative efforts were without
effect. Postmortem and toxicologic examinations revealed no apparent
cause for the death. Subsequent cardiologic evaluation of the
patient's 12-year-old sibling including echocardiography and
electrocardiography was within normal limits."

Source:

http://tinyurl.com/28ycon

To begin with, that's a story from 2002, already discussed to its full
extent.

There is no statute of limitations on homicide.

And where do you see the imperative connection between the tragic
death of that young lady and her diet?

The connection should be obvious to the most casual observer.

IMHO there's no prove at all.

There is no such thing as proof in science.

AFAICS there was no electrocardiography of that girl, so why shouldn't
it have been a hereditary Long QT syndrome or some other primary
electrophysiologic aberration?

An EKG was reportedly done during resuscitation efforts and on family
members.

I don't have to tell you, that the LQTS
is anything but a rare disease with an estimated prevalence of 1 in
10000 individuals.

Sudden cardiac death from LQTS in an otherwise healthy 16 year old
girl is rare.

It's thought to cause about 4000 deaths in the
United States each year, so a mere coincidence with a dietary measure
is anything but impossible. Or do you think the postmortal laboratory
parameters, which may be additionally influenced by infusions in the
course of the attempt of resuscitation, are qualified to supply any
relevant information? I don't.

In this casuistry the helpless attempts to explain the cause present a
mostly unrelated potpourri of more or less proven assertions leading
from gout via fatty acid induced arrhythmia in predamaged myocardium
and deaths caused by myocardial atrophy and myocarditis, all of which
weren't ascertainable in the course of the authopsy, up to a study
about liquid protein diet, which doesn't apply as well. Such a
strategy may be efficient in influencing the inclined reader, but in
principle is nothing but absolutely unscientific propaganda.

Suggested reading:

http://www.americanheart.org/present...entifier=11234

Be hungry... be healthy... be happy... be blessed:

http://TheWellnessFoundation.com/PressRelease

Prayerfully in Jesus' awesome love,

Andrew
--
Andrew B. Chung, MD/PhD
Cardiologist

neighbor Michael Sand wrote:
Andrew, in the Holy Spirit, boldly wrote:
neighbor Michael Sand wrote:

And where do you see the imperative connection between the tragic
death of that young lady and her diet?

The connection should be obvious to the most casual observer.

.. and vanishes if one digs deeper.

No it does not:

http://TruthRUS.org/lowcarb.pdf

IMHO there's no prove at all.

There is no such thing as proof in science.

So you question your own argumentation. Then what do you intend to
achieve with this casuistry?

This remains an advisory that started with the false claim that low-
carb dieting is associated with long life.

Obviously 16 years would not be a long life.

AFAICS there was no electrocardiography of that girl, so why shouldn't
it have been a hereditary Long QT syndrome or some other primary
electrophysiologic aberration?

An EKG was reportedly done during resuscitation efforts and on family
members.

During ventriculary fibrillation you get no information about
electrophysiologic irregularities.

Correct.

For this reason, a 12-lead EKG would not be done until after sinus
rhythm is restored with a defib shock.

Or what criteria of a LQTS would
you expect to find in such an ECG?

Think about it.

Moreover, it's not mandatory that
the ECG of LQTS patients' family members have to be pathologic.

Did not write that it were mandatory. However, the absence of EKG
abnormalities in family members makes it less likely that this 16-year-
old girl died from LQTS.

I don't have to tell you, that the LQTS
is anything but a rare disease with an estimated prevalence of 1 in
10000 individuals.

Sudden cardiac death from LQTS in an otherwise healthy 16 year old
girl is rare.

No, it's not.

If it were not, then it should be easy for you to cite a case of
sudden cardiac death from LQTS in an otherwise healthy 16 year old
girl that has happened this week.

The insidious characteristic of this disease are
syncopes and sudden death due to ventricular arrhythmias without any
prodromal symptoms.

Neither sudden cardiac death nor syncopes are insiduous.

It's thought to cause about 4000 deaths in the
United States each year, so a mere coincidence with a dietary measure
is anything but impossible. Or do you think the postmortal laboratory
parameters, which may be additionally influenced by infusions in the
course of the attempt of resuscitation, are qualified to supply any
relevant information? I don't.

In this casuistry the helpless attempts to explain the cause present a
mostly unrelated potpourri of more or less proven assertions leading
from gout via fatty acid induced arrhythmia in predamaged myocardium
and deaths caused by myocardial atrophy and myocarditis, all of which
weren't ascertainable in the course of the authopsy, up to a study
about liquid protein diet, which doesn't apply as well. Such a
strategy may be efficient in influencing the inclined reader, but in
principle is nothing but absolutely unscientific propaganda.

Suggested reading:

http://www.americanheart.org/present...entifier=11234

Where in this article do you see conclusive support for your thesis?
Please be more specific.

This remains an advisory.

It should be obvious to the most casual observer that not only is
there no evidence that low-carb dieting lengthens life-span, there
remains concerns that it shortens lifespan as it likely did for this
particular 16-year-old girl.

Be hungry... be healthy... be happy... be blessed:

http://TheWellnessFoundation.com/PressRelease

Prayerfully in Jesus' awesome love,

Andrew
--
Andrew B. Chung, MD/PhD
Cardiologist

neighbor Michael Sand wrote:
Andrew, in the Holy Spirit, boldlywrote:
neighbor Michael Sand wrote:
Andrew, in the Holy Spirit, boldly wrote:
neighbor Michael Sand wrote:

AFAICS there was no electrocardiography of that girl, so why shouldn't
it have been a hereditary Long QT syndrome or some other primary
electrophysiologic aberration?

An EKG was reportedly done during resuscitation efforts and on family
members.

During ventriculary fibrillation you get no information about
electrophysiologic irregularities.

Correct.

For this reason, a 12-lead EKG would not be done until after sinus
rhythm is restored with a defib shock.

Which they unfortunately didn't succeed in.

The lack of success was not due to misfortune but rather because of
the low-carb dieting:

"Our patient's sudden onset of cardiorespiratory arrest occurred
without an underlying cause identified by history or on postmortem
examination. Initial electrocardiogram revealed ventricular
fibrillation, an unusual presenting arrythmia in a 16-year-old
adolescent without underlying cardiac disease or electrolyte
disturbance. During resuscitation, with a pH of 6.89, the serum
potassium level was 3.8 mEq/L, suggesting profound hypokalemia if
corrected for the pH. The ionized calcium level was 1.12 mEq/L
(normal, 1.10 to 1.22 mEq/L). As with serum potassium, correction for
the pH would indicate significant hypocalcemia. Serum magnesium level
was not measured. Because potassium and magnesium undergo similar
handling mechanisms in the renal tubules, hypomagnesemia is another
potential cause of our patient's cardiorespiratory arrest. When
considering the potential causes of these electrolyte disturbances in
an otherwise healthy female adolescent, questions arise regarding the
potential role of the low-carbohydrate/high-protein diet compounded by
a period of inadequate caloric intake and the resultant catabolic
state."

Source:

http://www.medscape.com/viewarticle/442894_print

Or what criteria of a LQTS would
you expect to find in such an ECG?

Think about it.

I explicitly asked you, as you alleged to have a different POV.

I do not have a different POV from the doctors who attempted to
resuscitate the 16-year-old girl.

It seems you believe that they missed the diagnosis of LQTS.

Moreover, it's not mandatory that
the ECG of LQTS patients' family members have to be pathologic.

Did not write that it were mandatory. However, the absence of EKG
abnormalities in family members makes it less likely that this 16-year-
old girl died from LQTS.

I don't have to tell you, that the LQTS
is anything but a rare disease with an estimated prevalence of 1 in
10000 individuals.

Sudden cardiac death from LQTS in an otherwise healthy 16 year old
girl is rare.

No, it's not.

If it were not, then it should be easy for you to cite a case of
sudden cardiac death from LQTS in an otherwise healthy 16 year old
girl that has happened this week.

http://www.emedicine.com/med/topic1983.htm, written and edited by
leading cardiologists:

| It is important to review the ECGs of family members of a patient
| with LQTS, to obtain detail histories, and to perform physical
| examinations. However, an absence of ECG findings of LQTS in family
| members does not exclude LQTS.
|
| LQTS may result in syncope and lead to sudden cardiac death, which
| usually occurs in otherwise healthy young individuals. LQTS is
| thought to cause about 4000 deaths in the United States each year.
| The cumulative mortality rate reaches approximately 6% by the age of
| 40 years.
|
| Although sudden death usually occurs in symptomatic patients, it
| happens with the first episode of syncope in about 30% of the
| patients. This occurrence emphasizes the importance of diagnosing
| LQTS in the presymptomatic period.
|
| Patients with LQTS usually present with cardiac events (eg, syncope,
| aborted cardiac arrest, sudden death) in childhood, adolescence, or
| early adulthood. However, LQTS has been identified in adults as late
| as in the fifth decade of life. The risk of death from LQTS is
| higher in boys than in girls younger than 10 years, and the risk is
| similar in male and female patients thereafter.

Do not see where in your cite is it written that LQTS is a common
cause of death in otherwise healthy 16-year-old girls.

The insidious characteristic of this disease are
syncopes and sudden death due to ventricular arrhythmias without any
prodromal symptoms.

Neither sudden cardiac death nor syncopes are insiduous.

But the lack of prodromal signs is.

In the majority of folks with LQTS, there is no lack of prodromal
signs.

Be hungry... be healthy... be happy... be blessed:

http://TheWellnessFoundation.com/PressRelease

Prayerfully in Jesus' awesome love,

Andrew
--
Andrew B. Chung, MD/PhD
Cardiologist

neighbor Michael Sand wrote:
Andrew, in the Holy Spirit, boldly wrote:
neighbor Michael Sand wrote:
Andrew, in the Holy Spirit, boldly wrote:
neighbor Michael Sand wrote:

Or what criteria of a LQTS would
you expect to find in such an ECG?

Think about it.

I explicitly asked you, as you alleged to have a different POV.

I do not have a different POV from the doctors who attempted to
resuscitate the 16-year-old girl.

It seems you believe that they missed the diagnosis of LQTS.

It's obvious that they ignored alternative potential causes, one of
which LQTS is, and misuse this case to pursue their aims concerning
dieting.

LQTS does not explain the electrolyte abnormalities whereas low-carb
dieting does.

"Our patient's sudden onset of cardiorespiratory arrest occurred
without an underlying cause identified by history or on postmortem
examination. Initial electrocardiogram revealed ventricular
fibrillation, an unusual presenting arrythmia in a 16-year-old
adolescent without underlying cardiac disease or electrolyte
disturbance. During resuscitation, with a pH of 6.89, the serum
potassium level was 3.8 mEq/L, suggesting profound hypokalemia if
corrected for the pH. The ionized calcium level was 1.12 mEq/L
(normal, 1.10 to 1.22 mEq/L). As with serum potassium, correction for
the pH would indicate significant hypocalcemia. Serum magnesium level
was not measured. Because potassium and magnesium undergo similar
handling mechanisms in the renal tubules, hypomagnesemia is another
potential cause of our patient's cardiorespiratory arrest. When
considering the potential causes of these electrolyte disturbances in
an otherwise healthy female adolescent, questions arise regarding the
potential role of the low-carbohydrate/high-protein diet compounded
by
a period of inadequate caloric intake and the resultant catabolic
state."

Source:

http://www.medscape.com/viewarticle/442894_print

Truth is simple.

Be hungry... be healthy... be happy... be blessed:

http://TheWellnessFoundation.com/PressRelease

Prayerfully in Jesus' awesome love,

Andrew
--
Andrew B. Chung, MD/PhD
Cardiologist

neighbor Michael Sand wrote:
Andrew, in the Holy Spirit, boldly wrote:

LQTS does not explain the electrolyte abnormalities whereas low-carb
dieting does.

"Our patient's sudden onset of cardiorespiratory arrest occurred
without an underlying cause identified by history or on postmortem
examination. Initial electrocardiogram revealed ventricular
fibrillation, an unusual presenting arrythmia in a 16-year-old
adolescent without underlying cardiac disease or electrolyte
disturbance. During resuscitation, with a pH of 6.89, the serum
potassium level was 3.8 mEq/L, suggesting profound hypokalemia if
corrected for the pH. The ionized calcium level was 1.12 mEq/L
(normal, 1.10 to 1.22 mEq/L). As with serum potassium, correction for
the pH would indicate significant hypocalcemia. Serum magnesium level
was not measured. Because potassium and magnesium undergo similar
handling mechanisms in the renal tubules, hypomagnesemia is another
potential cause of our patient's cardiorespiratory arrest. When
considering the potential causes of these electrolyte disturbances in
an otherwise healthy female adolescent, questions arise regarding the
potential role of the low-carbohydrate/high-protein diet compounded
by a period of inadequate caloric intake and the resultant catabolic
state."

Source:

http://www.medscape.com/viewarticle/442894_print

Your allegedly irrefutable statements still base on superficial
knowledge.

The statements are not mine but those of the physicians who were given
the task of resuscitating this teenage girl. It would be my choice to
refrain from opining that their fund of knowledge is superficial.
Instead, the teenager's surviving friends and family will continue to
have my condolences for their loss.

Moreover, the opinion that these statements are irrefutable are your
own and not mine.

However, I would agree with your unwitting assessment that aside from
the teenager's physicians, we are in no position to refute their
clinical assessment without having been there to examine her and to
witness their resuscitation efforts. Therefore, their statements are
essentially irrefutable by us.

It should be noted that their conclusions that we should be concerned
about adverse outcomes from low-carb dieting is entirely consistent
with the application of Occam's Razor.

GOD remains the Author of all reality including the alternate ones.

For this reason, the brethren of LORD Jesus Christ in their walk with
HIM stay grounded in reality although the same can not be written
about those who remain of this world:

http://www.interviewwithgod.com/forgiven/

Be hungry... be healthy... be happy... be blessed:

http://TheWellnessFoundation.com/PressRelease

Prayerfully in Jesus' awesome love,

Andrew
--
Andrew B. Chung, MD/PhD
Cardiologist

On Aug 19, 2:22 pm, "Andrew B. Chung, MD/PhD"
wrote:

The statements are not mine but those of the physicians who were given
the task of resuscitating this teenage girl. It would be my choice to
refrain from opining that their fund of knowledge is superficial.
Instead, the teenager's surviving friends and family will continue to
have my condolences for their loss.

My GOD, Dr. Chung. Did you kill someone?