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Is aspartame really safer in reducing the risk of hypoglycemia during exercise in patients with type 2 diabetes? Ferland A, Brassard P, Poirier P, Universite Laval, Quebec, Diabetes Care 2007 July: Murray 2007.07.06
Is aspartame really safer in reducing the risk of hypoglycemia during
exercise in patients with type 2 diabetes? Ferland A, Brassard P, Poirier P, Universite Laval, Quebec, Diabetes Care 2007 July: Murray 2007.07.06 http://groups.yahoo.com/group/aspartameNMmessage/1450 " Although it has been reported that various forms of carbohydrate intake before exercise is safer for patients with diabetes in the prevention of exercise- induced hypoglycemia, we have reported severe symptoms of hypoglycemia on four occasions following sucrose - and/or aspartame sweetened meals (6). Considering the lack of evidence on the aspartame utilization in patients with type 2 diabetes (7-9), we consider that these clinical observations, in a exercise setting, raise important concerns regarding the safety of aspartame as suggested by international guidelines. " [ See also: aspartame and sucrose each raise blood glucose in diabetes 2, Annie Ferland, Paul Poirier, et al, talk 2005.05.13 Laval Hospital Research Center, Laval University, Sainte-Foy, Canada: Murray 2007.01.11 http://groups.yahoo.com/group/aspartameNM/message/1401 ] Diabetes Care. 2007 July; 30(7). page e59. Is aspartame really safer in reducing the risk of hypoglycemia during exercise in patients with type 2 diabetes? Ferland A, Brassard P, Poirier P. PMID: 17596482 OBSERVATIONS Is Aspartame Really Safer in Reducing the Risk of Hypoglycemia During Exercise in Patients With Type 2 Diabetes? In addition to physical activity and healthy food choices, low-calorie sweetening agents, such as aspartame, are a recommended alternative to sugar for patients with type 2 diabetes in order to obtain a better control of carbohydrate intake and blood glucose levels (1-3). The safety of aspartame has been a controversial issue for quite some time now. This noncarbohydrate sweetener is currently found in over 6,000 food products and beverages throughout the world. At present, its attractiveness as an artificial sweetener in the dietary management of diabetes is related to its about 00-fold sweetening power and the lack of effect on plasma glucose levels compared with sucrose. We have recently investigated the effect of different macronutrient compositions on plasma glucose and insulin levels during an acute bout of exercise in 14 men with type 2 diabetes. We compared the same subjects in random order in five different conditions: 1) high- glycemic index sucrose meal, 2) low-glycemic index fructose meal (both of which are matched for total calories [455 kcal], macronutrient composition, and taste), 3) aspartame meal (358 kcal), 4) high-fat/lowcarbohydrate meal (also containing 455 kcal), and 5) fasting. We hypothesized that using fructose or aspartame instead of sucrose would have a lower impact on insulin release and glucose response than a sucrose-sweetened meal. Contrary to all expectation, the aspartame breakfast induced a similar rise in glucose and insulin levels at baseline than the sucrose meal, even if the aspartame meal had the same taste, and was 22% lower in calories and 10% lower in carbohydrates, with an inferior glycemic index. Indeed, the most dramatic reduction in plasma glucose level occurred in those with the highest 2-h postprandial plasma glucose levels ( over 8 mmol/ l ), i.e., after the sucrose, high-fat/low-carbohydrate and aspartame meals (magnitude of decrease of 44, 37, and 34%, respectively; all P 0.001). However, the fructose meal induced the lowest fall in blood glucose, with a 31% decrease from baseline. An important fall was also observed in plasma insulin levels (78, 75, 73, and 68% for the sucrose, high-fat/lowcarbohydrate, aspartame, and fructose meals, respectively; all P 0.001), in contrast to the fasting state where there were no changes. Moreover, considering that all sweetened meals (sucrose, fructose, and aspartame) in our study had the same taste, we can speculate that aspartame might have enhanced the cephalic phase of insulin secretion evoked by the recognition of the sweet taste, sight, smell, and expectation of food (4,5) and could have potentiated the drop in glucose levels during exercise. Although it has been reported that various forms of carbohydrate intake before exercise is safer for patients with diabetes in the prevention of exercise- induced hypoglycemia, we have reported severe symptoms of hypoglycemia on four occasions following sucrose - and/or aspartame sweetened meals (6). Considering the lack of evidence on the aspartame utilization in patients with type 2 diabetes (7-9), we consider that these clinical observations, in a exercise setting, raise important concerns regarding the safety of aspartame as suggested by international guidelines. ANNIE FERLAND, RD, MSC PATRICE BRASSARD, MSC PAUL POIRIER, MD, PHD, FRCPC, FACC, FAHA From the Institut Universitaire de Cardiologie et de Pneumologie, Hospital Laval, UniversiteŽ Laval, Sainte-Foy, QueŽbec, Canada. Address correspondence to Paul Poirier, MD, PhD, FRCPC, FACC, FAHA, Institut Universitaire de Cardiologie et de Pneumologie, Hospital Laval, 2725 Chemin Sainte-Foy, Sainte-Foy, QueŽbec, Canada G1V 4G5. E-mail: DOI: 10.2337/dc06-1888 © 2007 by the American Diabetes Association. Acknowledgments-This work was supported by the Canadian Diabetes Association and the Foundation of the Corporation of the Quebec Heart Institute. A.F. is supported by the Canadian Institutes of Health Research (CIHR). P.B. is the recipient of a graduate research scholarship in pharmacy (PhD) from the Rx & D Health Research Foundation Awards Program funded in partnership with the CIHR. P.P. is a Clinician Scientist of the Fonds de la Recherche en SanteŽ du QueŽbec. References 1. American Diabetes Association: Standards of medical care in diabetes - 2006. Diabetes Care 29 (Suppl. 1): S4-S42, 2006 2. Gougeon R, Spidel M, Lee K, Field CJ: Canadian Diabetes Association National Nutrition Committee Technical Review: non-nutritive intense sweeteners in diabetes management. Can J Diabetes 28: 385-399, 2004 3. Canadian Diabetes Association: Clinical practice guidelines for the prevention and management of diabetes in Canada. Can J Diabetes 27 (Suppl. 2): 1-163, 2003 4. Bruce DG, Storlien LH, Furler SM, Chisholm DJ: Cephalic phase metabolic responses in normal weight adults. Metabolism 36: 721-725, 1987 5. Melanson KJ, Westerterp-Plantenga MS, Campfield LA, Saris WH: Blood glucose and meal patterns in time-blinded males, after aspartame, carbohydrate, and fat consumption, in relation to sweetness perception. Br J Nutr 82: 437 - 446, 1999 6. Ferland A, Turbide G, Bogaty P, Jobin J, Larivie`re MM, Poirier P: Type 2 diabetes and aerobic exercise: is breakfast deleterious (Abstract)? Med Sci Sports Exerc 36: S330, 2004 7. Colagiuri S, Miller JJ, Edwards RA: Metabolic effects of adding sucrose and aspartame to the diet of subjects with noninsulin-dependent diabetes mellitus. Am J Clin Nutr 50: 474-478, 1989 8. Horwitz DL, McLane M, Kobe P: Response to single dose of aspartame or saccharin by NIDDM patients. Diabetes Care 11: 230-234, 1988 9. Shigeta H, Yoshida T, Nakai M, Mori H, Kano Y, Nishioka H, Kajiyama S, Kitagawa Y, Kanatsuna T, Kondo M, et al.: Effects of aspartame on diabetic rats and diabetic patients. Nutr Sci Vitaminol (Tokyo) 31: 533-540, 1985 ONLINE LETTERS DIABETES CARE, VOLUME 30, NUMBER 7, JULY 2007 page e59 ////////////////////////////////////////////////////////// "Of course, everyone chooses, as a natural priority, to actively find, quickly share, and positively act upon the facts about healthy and safe food, drink, and environment." Rich Murray, MA Room For All 505-501-2298 1943 Otowi Road, Santa Fe, New Mexico 87505 http://groups.yahoo.com/group/aspartameNM/messages group with 77 members, 1,450 posts in a public, searchable archive http://RMForAll.blogspot.com http://groups.yahoo.com/group/aspartameNMmessage/1443 Safe Food Campaign wants ban on aspartame in schools in New Zealand: Murray 2007.06.21 http://groups.yahoo.com/group/aspartameNM/message/1442 Wellington, NZ lady, 25, free by 24 hours of severe muscle cramps (5 months) after quitting 4-8 packs daily aspartame chewing gum (past few years): Murray 2007.06.20 http://groups.yahoo.com/group/aspartameNM/message/1441 Lifetime exposure to low doses of aspartame beginning during prenatal life increases cancer effects in rats, Morando Soffritti et al, European Ramazzini Foundation, USA EPA Environmental Health Perspectives 2007.06.13 free full text 24 pages: Murray 2007.06.16 www.ehponline.org/members/2007/10271/10271.pdf free full text 24 pages http://groups.yahoo.com/group/aspartameNMmessage/1437 stevia to be approved and cyclamates limited by Food Standards Australia New Zealand: JMC Geuns critiques of two recent stevia studies by Nunes: Murray 2007.05.29 http://groups.yahoo.com/group/aspartameNM/message/1427 more from The Independent, UK, Martin Hickman, re ASDA (unit of Wal-Mart Stores) and Marks & Spencer ban of aspartame, MSG, artificial chemical additives and dyes to prevent ADHD in kids: Murray 2007.05.16 http://news.independent.co.uk/uk/hea...cle2548747.ece http://groups.yahoo.com/group/aspartameNM/message/1426 ASDA (unit of Wal-Mart Stores WMT.N) and Marks & Spencer will join Tesco and also Sainsbury to ban and limit aspartame, MSG, artificial flavors dyes preservatives additives, trans fats, salt "nasties" to protect kids from ADHD: leading UK media: Murray 2007.05.15 http://groups.yahoo.com/group/aspartameNM/message/1271 combining aspartame and quinoline yellow, or MSG and brilliant blue, harms nerve cells, eminent C. Vyvyan Howard et al, 2005 education.guardian.co.uk, Felicity Lawrence: Murray 2005.12.21 http://groups.yahoo.com/group/aspartameNM/message/1277 50% UK baby food is now organic -- aspartame or MSG with food dyes harm nerve cells, CV Howard 3 year study funded by Lizzy Vann, CEO, Organix Brands, Children's Food Advisory Service: Murray 2006.01.13 formaldehyde as a potent unexamined cofactor in cancer research -- sources include methanol, dark wines and liquors, aspartame, wood and tobacco smoke: IARC Monographs on the Evaluation of Carcinogenic Risks to Humans implicate formaldehyde in #88 and alcohol drinks in #96: some related abstracts: Murray 2007.05.15 http://groups.yahoo.com/group/aspartameNM/message/1417 aspartame (methanol, formaldehyde) toxicity research summary: Rich Murray 2007.06.16 http://groups.yahoo.com/group/aspartameNM/message/1404 One liter aspartame diet soda, about 3 12-oz cans, gives 61.5 mg methanol, so if 30% is turned into formaldehyde, the formaldehyde dose of 18.5 mg is 37 times the recent EPA limit of 0.5 mg per liter daily drinking water for a 10-kg child: http://www.epa.gov/teach/chem_summ/F...de_summary.pdf 2007.01.05 [ does not discuss formaldehyde from methanol or aspartame ] http://www.epa.gov/teach/teachsurvey.html comments http://groups.yahoo.com/group/aspartameNM/message/1340 aspartame groups and books: updated research review of 2004.07.16: Murray 2006.05.11 http://groups.yahoo.com/group/aspartameNM/message/1395 Aspartame Controversy, in Wikipedia democratic encyclopedia, 72 references (including AspartameNM # 864 and 1173 by Murray), brief fair summary of much more research: Murray 2007.01.01 Dark wines and liquors, as well as aspartame, provide similar levels of methanol, above 120 mg daily, for long-term heavy users, 2 L daily, about 6 cans. Within hours, methanol is inevitably largely turned into formaldehyde, and thence largely into formic acid -- the major causes of the dreaded symptoms of "next morning" hangover. Fully 11% of aspartame is methanol -- 1,120 mg aspartame in 2 L diet soda, almost six 12-oz cans, gives 123 mg methanol (wood alcohol). If 30% of the methanol is turned into formaldehyde, the amount of formaldehyde, 37 mg, is 18.5 times the USA EPA limit for daily formaldehyde in drinking water, 2.0 mg in 2 L average daily drinking water. http://groups.yahoo.com/group/aspartameNM/message/1286 methanol products (formaldehyde and formic acid) are main cause of alcohol hangover symptoms [same as from similar amounts of methanol, the 11% part of aspartame]: YS Woo et al, 2005 Dec: Murray 2006.01.20 http://groups.yahoo.com/group/aspartameNM/message/1143 methanol (formaldehyde, formic acid) disposition: Bouchard M et al, full plain text, 2001: substantial sources are degradation of fruit pectins, liquors, aspartame, smoke: Murray 2005.04.02 http://groups.yahoo.com/group/aspartameNMmessage/1447 second study by expert Greek team of neurotoxicity in infant rats by aspartame (or its parts, methanol, phenylalanine, aspartic acid), KH Schulpis et al, Toxicology 2007.05.18: Murray 2007.07.04 http://groups.yahoo.com/group/aspartameNMmessage/1444 expert Greek group finds aspartame (or its parts, methanol, phenylalanine, aspartic acid) harm infant rat brain enzyme activity, KH Schulpis et al, Pharmacol. Res. 2007.05.13: Murray 2007.06.23 http://groups.yahoo.com/group/aspartameNMmessage/1448 Sweet Misery -- A Poisoned World, free full 90 minute video on aspartame toxicity, Cori Brackett, Sound and Fury Productions Inc., video.google.com: Murray 2007.07.04 ////////////////////////////////////////////////////////// |
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Is aspartame really safer in reducing the risk of hypoglycemia during exercise in patients with type 2 diabetes? Ferland A, Brassard P, Poirier P, Universite Laval, Quebec, Diabetes Care 2007 July: Murray 2007.07.06
"Rich Murray" wrote in message
ups.com... Is aspartame really safer in reducing the risk of hypoglycemia during exercise in patients with type 2 diabetes? Well, you sure could not tell from this study. The cephalic response is what is in question here. This response is well known, but the question is whether and to what extent it occurs when food sweetened with artificial sweeteners is eaten. The quoted study suggests that yes, it may occur with foods sweetened with aspartame. The result is that the body secretes more insulin than necessary based on the artifically sweet taste, resulting in some degree of lowered blood glucose. The problem is, I believe studies demonstrating a cephalic insulin response have not been consistent. Even using the same artificial sweetener. They also have been very short term, so far as I'm aware, so whether or not the body learns over time to adjust insulin response following intake of artificial sweeteners has not been researched adequately. Personally, I suspect that there is some adjustment. When I first started lc'ing, years back, I could bring on a nice hypo episode by drinking a single Diet Coke, especially between meals. That stopped happening after a few weeks. I don't know the reason for the change. Maybe my blood sugar levels just became more stable and a little ding one way or the other didn't impact how I felt. Or possibly my body learned to adjust insulin output more appropriately when I'd ingested a non-caloric sweetener. Definitely room for more research here (needs to be longer term, however.) HG |
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Is aspartame really safer in reducing the risk of hypoglycemia during exercise in patients with type 2 diabetes? Ferland A, Brassard P, Poirier P, Universite Laval, Quebec, Diabetes Care 2007 July: Murray 2007.07.06
On 6 Jul, 23:38, Rich Murray wrote:
Is aspartame really safer in reducing the risk of hypoglycemia during exercise in patients with type 2 diabetes? Ferland A, Brassard P, Poirier P, Universite Laval, Quebec, Diabetes Care 2007 July: Murray 2007.07.06http://groups.yahoo.com/group/aspartameNMmessage/1450 " Although it has been reported that various forms of carbohydrate intake before exercise is safer for patients with diabetes in the prevention of exercise- induced hypoglycemia, we have reported severe symptoms of hypoglycemia on four occasions following sucrose - and/or aspartame sweetened meals (6). Considering the lack of evidence on the aspartame utilization in patients with type 2 diabetes (7-9), we consider that these clinical observations, in a exercise setting, raise important concerns regarding the safety of aspartame as suggested by international guidelines. " [ See also: aspartame and sucrose each raise blood glucose in diabetes 2, Annie Ferland, Paul Poirier, et al, talk 2005.05.13 Laval Hospital Research Center, Laval University, Sainte-Foy, Canada: Murray 2007.01.11http://groups.yahoo.com/group/aspartameNM/message/1401] Diabetes Care. 2007 July; 30(7). page e59. Is aspartame really safer in reducing the risk of hypoglycemia during exercise in patients with type 2 diabetes? Ferland A, Brassard P, Poirier P. PMID: 17596482 OBSERVATIONS Is Aspartame Really Safer in Reducing the Risk of Hypoglycemia During Exercise in Patients With Type 2 Diabetes? In addition to physical activity and healthy food choices, low-calorie sweetening agents, such as aspartame, are a recommended alternative to sugar for patients with type 2 diabetes in order to obtain a better control of carbohydrate intake and blood glucose levels (1-3). The safety of aspartame has been a controversial issue for quite some time now. This noncarbohydrate sweetener is currently found in over 6,000 food products and beverages throughout the world. At present, its attractiveness as an artificial sweetener in the dietary management of diabetes is related to its about 00-fold sweetening power and the lack of effect on plasma glucose levels compared with sucrose. We have recently investigated the effect of different macronutrient compositions on plasma glucose and insulin levels during an acute bout of exercise in 14 men with type 2 diabetes. We compared the same subjects in random order in five different conditions: 1) high- glycemic index sucrose meal, 2) low-glycemic index fructose meal (both of which are matched for total calories [455 kcal], macronutrient composition, and taste), 3) aspartame meal (358 kcal), 4) high-fat/lowcarbohydrate meal (also containing 455 kcal), and 5) fasting. We hypothesized that using fructose or aspartame instead of sucrose would have a lower impact on insulin release and glucose response than a sucrose-sweetened meal. Contrary to all expectation, the aspartame breakfast induced a similar rise in glucose and insulin levels at baseline than the sucrose meal, even if the aspartame meal had the same taste, and was 22% lower in calories and 10% lower in carbohydrates, with an inferior glycemic index. Indeed, the most dramatic reduction in plasma glucose level occurred in those with the highest 2-h postprandial plasma glucose levels ( over 8 mmol/ l ), i.e., after the sucrose, high-fat/low-carbohydrate and aspartame meals (magnitude of decrease of 44, 37, and 34%, respectively; all P 0.001). However, the fructose meal induced the lowest fall in blood glucose, with a 31% decrease from baseline. An important fall was also observed in plasma insulin levels (78, 75, 73, and 68% for the sucrose, high-fat/lowcarbohydrate, aspartame, and fructose meals, respectively; all P 0.001), in contrast to the fasting state where there were no changes. Moreover, considering that all sweetened meals (sucrose, fructose, and aspartame) in our study had the same taste, we can speculate that aspartame might have enhanced the cephalic phase of insulin secretion evoked by the recognition of the sweet taste, sight, smell, and expectation of food (4,5) and could have potentiated the drop in glucose levels during exercise. Although it has been reported that various forms of carbohydrate intake before exercise is safer for patients with diabetes in the prevention of exercise- induced hypoglycemia, we have reported severe symptoms of hypoglycemia on four occasions following sucrose - and/or aspartame sweetened meals (6). Considering the lack of evidence on the aspartame utilization in patients with type 2 diabetes (7-9), we consider that these clinical observations, in a exercise setting, raise important concerns regarding the safety of aspartame as suggested by international guidelines. ANNIE FERLAND, RD, MSC PATRICE BRASSARD, MSC PAUL POIRIER, MD, PHD, FRCPC, FACC, FAHAFrom the Institut Universitaire de Cardiologie et de Pneumologie, Hospital Laval, UniversiteŽ Laval, Sainte-Foy, QueŽbec, Canada. Address correspondence to Paul Poirier, MD, PhD, FRCPC, FACC, FAHA, Institut Universitaire de Cardiologie et de Pneumologie, Hospital Laval, 2725 Chemin Sainte-Foy, Sainte-Foy, QueŽbec, Canada G1V 4G5. E-mail: DOI: 10.2337/dc06-1888 © 2007 by the American Diabetes Association. Acknowledgments-This work was supported by the Canadian Diabetes Association and the Foundation of the Corporation of the Quebec Heart Institute. A.F. is supported by the Canadian Institutes of Health Research (CIHR). P.B. is the recipient of a graduate research scholarship in pharmacy (PhD) from the Rx & D Health Research Foundation Awards Program funded in partnership with the CIHR. P.P. is a Clinician Scientist of the Fonds de la Recherche en SanteŽ du QueŽbec. References 1. American Diabetes Association: Standards of medical care in diabetes - 2006. Diabetes Care 29 (Suppl. 1): S4-S42, 2006 2. Gougeon R, Spidel M, Lee K, Field CJ: Canadian Diabetes Association National Nutrition Committee Technical Review: non-nutritive intense sweeteners in diabetes management. Can J Diabetes 28: 385-399, 2004 3. Canadian Diabetes Association: Clinical practice guidelines for the prevention and management of diabetes in Canada. Can J Diabetes 27 (Suppl. 2): 1-163, 2003 4. Bruce DG, Storlien LH, Furler SM, Chisholm DJ: Cephalic phase metabolic responses in normal weight adults. Metabolism 36: 721-725, 1987 5. Melanson KJ, Westerterp-Plantenga MS, Campfield LA, Saris WH: Blood glucose and meal patterns in time-blinded males, after aspartame, carbohydrate, and fat consumption, in relation to sweetness perception. Br J Nutr 82: 437 - 446, 1999 6. Ferland A, Turbide G, Bogaty P, Jobin J, Larivie`re MM, Poirier P: Type 2 diabetes and aerobic exercise: is breakfast deleterious (Abstract)? Med Sci Sports Exerc 36: S330, 2004 7. Colagiuri S, Miller JJ, Edwards RA: Metabolic effects of adding sucrose and aspartame to the diet of subjects with noninsulin-dependent diabetes mellitus. Am J Clin Nutr 50: 474-478, 1989 8. Horwitz DL, McLane M, Kobe P: Response to single dose of aspartame or saccharin by NIDDM patients. Diabetes Care 11: 230-234, 1988 9. Shigeta H, Yoshida T, Nakai M, Mori H, Kano Y, Nishioka H, Kajiyama S, Kitagawa Y, Kanatsuna T, Kondo M, et al.: Effects of aspartame on diabetic rats and diabetic patients. Nutr Sci Vitaminol (Tokyo) 31: 533-540, 1985 ONLINE LETTERS DIABETES CARE, VOLUME 30, NUMBER 7, JULY 2007 page e59 ////////////////////////////////////////////////////////// "Of course, everyone chooses, as a natural priority, to actively find, quickly share, and positively act upon the facts about healthy and safe food, drink, and environment." Rich Murray, MA Room For All 505-501-2298 1943 Otowi Road, Santa Fe, New Mexico 87505 http://groups.yahoo.com/group/aspartameNM/messages group with 77 members, 1,450 posts in a public, searchable archivehttp://RMForAll.blogspot.com http://groups.yahoo.com/group/aspartameNMmessage/1443 Safe Food Campaign wants ban on aspartame in schools in New Zealand: Murray 2007.06.21 http://groups.yahoo.com/group/aspartameNM/message/1442 Wellington, NZ lady, 25, free by 24 hours of severe muscle cramps (5 months) after quitting 4-8 packs daily aspartame chewing gum (past few years): Murray 2007.06.20 http://groups.yahoo.com/group/aspartameNM/message/1441 Lifetime exposure to low doses of aspartame beginning during prenatal life increases cancer effects in rats, Morando Soffritti et al, European Ramazzini Foundation, USA EPA Environmental Health Perspectives 2007.06.13 free full text 24 pages: Murray 2007.06.16 www.ehponline.org/members/2007/10271/10271.pdf free full text 24 pages http://groups.yahoo.com/group/aspartameNMmessage/1437 stevia to be approved and cyclamates limited by Food Standards Australia New Zealand: JMC Geuns critiques of two recent stevia studies by Nunes: Murray 2007.05.29 http://groups.yahoo.com/group/aspartameNM/message/1427 more from The Independent, UK, Martin Hickman, re ASDA (unit of Wal-Mart Stores) and Marks & Spencer ban of aspartame, MSG, artificial chemical additives and dyes to prevent ADHD in kids: Murray 2007.05.16http://news.independent.co.uk/uk/health_medical/article2548747.ece http://groups.yahoo.com/group/aspartameNM/message/1426 ASDA (unit of Wal-Mart Stores WMT.N) and Marks & Spencer will join Tesco and also Sainsbury to ban and limit aspartame, MSG, artificial flavors dyes preservatives additives, trans fats, salt "nasties" to protect kids from ADHD: leading UK media: Murray 2007.05.15 http://groups.yahoo.com/group/aspartameNM/message/1271 combining aspartame and quinoline yellow, or MSG and brilliant blue, harms nerve cells, eminent C. Vyvyan Howard et al, 2005 education.guardian.co.uk, Felicity Lawrence: Murray 2005.12.21 http://groups.yahoo.com/group/aspartameNM/message/1277 50% UK baby food is now organic -- aspartame or MSG with food dyes harm nerve cells, CV Howard 3 year study funded by Lizzy Vann, CEO, Organix Brands, Children's ... read more » I come across information on the internet that aspartame is toxic and should be avoided. e.g. water is better than a diet drink. There are conspiracy theories as to how is was approved. |
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Is aspartame really safer in reducing the risk of hypoglycemia during exercise in patients with type 2 diabetes? Ferland A, Brassard P, Poirier P, Universite Laval, Quebec, Diabetes Care 2007 July: Murray 2007.07.06
"Dave" I come across information on the internet that aspartame is toxic and should be avoided. e.g. water is better than a diet drink. There are conspiracy theories as to how is was approved. You also reposted all of the original post only to add 2 sentences at the bottom which are idiotic. Therefore, you are an idiot. plonk |
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