Diabetes and Glycogen?

convicted neighbor Kumar wrote:

"Disorders of glycogen metabolism
The most common disease in which glycogen metabolism becomes abnormal
is diabetes, in which, because of abnormal amounts of insulin, liver
glycogen can be abnormally accumulated or depleted. Restoration of
normal glucose metabolism usually normalizes glycogen metabolism as
well.

In hypoglycemia caused by excessive insulin, liver glycogen levels are
high, but the high insulin level prevents the glycogenolysis necessary
to maintain normal blood sugar levels. Glucagon is a common treatment
for this type of hypoglycemia.
http://en.wikipedia.org/wiki/Glycogen "

Hunger is a feeling experienced when the glycogen level of the liver
falls below a threshold, usually followed by a desire to eat. The
usually unpleasant feeling originates in the hypothalamus and is
released through receptors in the liver.
http://en.wikipedia.org/wiki/Hunger "

If this were true, folks who are low-carbing would be hungrier but
they are not.

Hello,

Above look quite indicative of getting persistent hyperglycemia and
getting hunger in diabetes type2 ( due to indicated glycogen
abnormalities).

Hunger does not arise from a lack but from anticipation.

Simply observe how much hungrier you are when you smell something
wonderful cooking in the kitchen.

Why it can't be related to persisting hyperglycemia, insulin
resistance and excessive hunger/overeating in diabetes2?

Hunger is simply a healthy appetite.

"Hunger is good." -- Holy Spirit

Amen.

"Blessed are you who hunger now for you will be satisfied." -- LORD
Jesus Christ (Luke 6:21)

Amen ! Laus Deo ! ! ! Maranatha ! ! ! ! ! ! !

Andrew
--
Andrew B. Chung, MD/PhD
http://EmoryCardiology.com

On Feb 17, 7:06 am, "Andrew B. Chung, MD/PhD"
wrote:
convicted neighbor Kumar wrote:

"Disorders of glycogen metabolism
The most common disease in which glycogen metabolism becomes abnormal
is diabetes, in which, because of abnormal amounts of insulin, liver
glycogen can be abnormally accumulated or depleted. Restoration of
normal glucose metabolism usually normalizes glycogen metabolism as
well.

In hypoglycemia caused by excessive insulin, liver glycogen levels are
high, but the high insulin level prevents the glycogenolysis necessary
to maintain normal blood sugar levels. Glucagon is a common treatment
for this type of hypoglycemia.
http://en.wikipedia.org/wiki/Glycogen"

Hunger is a feeling experienced when the glycogen level of the liver
falls below a threshold, usually followed by a desire to eat. The
usually unpleasant feeling originates in the hypothalamus and is
released through receptors in the liver.
http://en.wikipedia.org/wiki/Hunger"

If this were true, folks who are low-carbing would be hungrier but
they are not.
Is it correct and healthful to manipulate sysnthesis or energy stores
and resist/restrict their breakdown by added insulin in diabetics2?
Hello,

Above look quite indicative of getting persistent hyperglycemia and
getting hunger in diabetes type2 ( due to indicated glycogen
abnormalities).

Hunger does not arise from a lack but from anticipation.

Simply observe how much hungrier you are when you smell something
wonderful cooking in the kitchen.
That can be due to cephalic phase effect and palatability/likings?
Few may get averse reactions on smelling unliked/undesired or not
needed foods
Why it can't be related to persisting hyperglycemia, insulin
resistance and excessive hunger/overeating in diabetes2?

Hunger is simply a healthy appetite.

"Hunger is good." -- Holy Spirit
Yes, but excess of everything is also considered bad. One can either
get real hunger on which you enjoy eatings as if some good deed is
done Whereas other representing backing of any disorder can be
apparant/not real which you don't enjoy and followed by guilt..as sin
is commited. Diabetics commonly experiances both.
Amen.

"Blessed are you who hunger now for you will be satisfied." -- LORD
Jesus Christ (Luke 6:21)

Amen ! Laus Deo ! ! ! Maranatha ! ! ! ! ! ! !

Andrew
--
Andrew B. Chung, MD/PhDhttp://EmoryCardiology.com- Hide quoted text -

- Show quoted text -

convicted neighbor kumar wrote:
Andrew, in the Holy Spirit, boldly wrote:
convicted neighbor Kumar wrote:

"Disorders of glycogen metabolism
The most common disease in which glycogen metabolism becomes abnormal
is diabetes, in which, because of abnormal amounts of insulin, liver
glycogen can be abnormally accumulated or depleted. Restoration of
normal glucose metabolism usually normalizes glycogen metabolism as
well.

In hypoglycemia caused by excessive insulin, liver glycogen levels are
high, but the high insulin level prevents the glycogenolysis necessary
to maintain normal blood sugar levels. Glucagon is a common treatment
for this type of hypoglycemia.
http://en.wikipedia.org/wiki/Glycogen"

Hunger is a feeling experienced when the glycogen level of the liver
falls below a threshold, usually followed by a desire to eat. The
usually unpleasant feeling originates in the hypothalamus and is
released through receptors in the liver.
http://en.wikipedia.org/wiki/Hunger"

If this were true, folks who are low-carbing would be hungrier but
they are not.

Is it correct and healthful to manipulate sysnthesis or energy stores
and resist/restrict their breakdown by added insulin in diabetics2?

The reason for administering insulin to a type-2 diabetic is simply to
lower blood glucose.

Hello,

Above look quite indicative of getting persistent hyperglycemia and
getting hunger in diabetes type2 ( due to indicated glycogen
abnormalities).

Hunger does not arise from a lack but from anticipation.

Simply observe how much hungrier you are when you smell something
wonderful cooking in the kitchen.

That can be due to cephalic phase effect and palatability/likings?

It is from anticipation.

Few may get averse reactions on smelling unliked/undesired or not
needed foods

No one needs chocolate chip cookies to survive yet nearly invariably
healthy folks get hungrier when they smell them baking in the kitchen
oven.

Why it can't be related to persisting hyperglycemia, insulin
resistance and excessive hunger/overeating in diabetes2?

Hunger is simply a healthy appetite.

"Hunger is good." -- Holy Spirit

Amen.

Yes, but excess of everything is also considered bad.

That would be the sin of gluttony regarding food.

One can either
get real hunger on which you enjoy eatings as if some good deed is
done Whereas other representing backing of any disorder can be
apparant/not real which you don't enjoy and followed by guilt..as sin
is commited. Diabetics commonly experiances both.

Not those who know in the heart of their soul that "hunger is good."

"Blessed are you who hunger now for you will be satisfied." -- LORD
Jesus Christ (Luke 6:21)

Amen ! Laus Deo ! ! ! Maranatha ! ! ! ! ! ! !

Andrew
--
Andrew B. Chung, MD/PhD
http://EmoryCardiology.com

On Feb 22, 7:43 pm, "Andrew B. Chung, MD/PhD"
wrote:
convicted neighbor kumar wrote:
Andrew, in the Holy Spirit, boldly wrote:
convicted neighbor Kumar wrote:

"Disorders of glycogen metabolism
The most common disease in which glycogen metabolism becomes abnormal
is diabetes, in which, because of abnormal amounts of insulin, liver
glycogen can be abnormally accumulated or depleted. Restoration of
normal glucose metabolism usually normalizes glycogen metabolism as
well.

In hypoglycemia caused by excessive insulin, liver glycogen levels are
high, but the high insulin level prevents the glycogenolysis necessary
to maintain normal blood sugar levels. Glucagon is a common treatment
for this type of hypoglycemia.
http://en.wikipedia.org/wiki/Glycogen"

Hunger is a feeling experienced when the glycogen level of the liver
falls below a threshold, usually followed by a desire to eat. The
usually unpleasant feeling originates in the hypothalamus and is
released through receptors in the liver.
http://en.wikipedia.org/wiki/Hunger"

If this were true, folks who are low-carbing would be hungrier but
they are not.

Is it correct and healthful to manipulate sysnthesis or energy stores
and resist/restrict their breakdown by added insulin in diabetics2?

The reason for administering insulin to a type-2 diabetic is simply to
lower blood glucose.
Which energy store need oxidation process?
Hello,

Above look quite indicative of getting persistent hyperglycemia and
getting hunger in diabetes type2 ( due to indicated glycogen
abnormalities).

Hunger does not arise from a lack but from anticipation.

Simply observe how much hungrier you are when you smell something
wonderful cooking in the kitchen.

That can be due to cephalic phase effect and palatability/likings?

It is from anticipation.

Few may get averse reactions on smelling unliked/undesired or not
needed foods

No one needs chocolate chip cookies to survive yet nearly invariably
healthy folks get hungrier when they smell them baking in the kitchen
oven.

Why it can't be related to persisting hyperglycemia, insulin
resistance and excessive hunger/overeating in diabetes2?

Hunger is simply a healthy appetite.

"Hunger is good." -- Holy Spirit

Amen.

Yes, but excess of everything is also considered bad.

That would be the sin of gluttony regarding food.

One can either
get real hunger on which you enjoy eatings as if some good deed is
done Whereas other representing backing of any disorder can be
apparant/not real which you don't enjoy and followed by guilt..as sin
is commited. Diabetics commonly experiances both.

Not those who know in the heart of their soul that "hunger is good."

"Blessed are you who hunger now for you will be satisfied." -- LORD
Jesus Christ (Luke 6:21)

Amen ! Laus Deo ! ! ! Maranatha ! ! ! ! ! ! !

Andrew
--
Andrew B. Chung, MD/PhDhttp://EmoryCardiology.com- Hide quoted text -

- Show quoted text -

convicted neighbor Kumar wrote:
Andrew, in the Holy Spirit, boldly wrote:
convicted neighbor kumar wrote:
Andrew, in the Holy Spirit, boldly wrote:
convicted neighbor Kumar wrote:

"Disorders of glycogen metabolism
The most common disease in which glycogen metabolism becomes abnormal
is diabetes, in which, because of abnormal amounts of insulin, liver
glycogen can be abnormally accumulated or depleted. Restoration of
normal glucose metabolism usually normalizes glycogen metabolism as
well.

In hypoglycemia caused by excessive insulin, liver glycogen levels are
high, but the high insulin level prevents the glycogenolysis necessary
to maintain normal blood sugar levels. Glucagon is a common treatment
for this type of hypoglycemia.
http://en.wikipedia.org/wiki/Glycogen"

Hunger is a feeling experienced when the glycogen level of the liver
falls below a threshold, usually followed by a desire to eat. The
usually unpleasant feeling originates in the hypothalamus and is
released through receptors in the liver.
http://en.wikipedia.org/wiki/Hunger"

If this were true, folks who are low-carbing would be hungrier but
they are not.

Is it correct and healthful to manipulate sysnthesis or energy stores
and resist/restrict their breakdown by added insulin in diabetics2?

The reason for administering insulin to a type-2 diabetic is simply to
lower blood glucose.

Which energy store need oxidation process?

Without oxygen for oxidation, you would die.

Andrew
--
Andrew B. Chung, MD/PhD
http://EmoryCardiology.com

On Feb 25, 12:15 am, "Andrew B. Chung, MD/PhD"
wrote:
convicted neighbor Kumar wrote:
Andrew, in the Holy Spirit, boldly wrote:
convicted neighbor kumar wrote:
Andrew, in the Holy Spirit, boldly wrote:
convicted neighbor Kumar wrote:

"Disorders of glycogen metabolism
The most common disease in which glycogen metabolism becomes abnormal
is diabetes, in which, because of abnormal amounts of insulin, liver
glycogen can be abnormally accumulated or depleted. Restoration of
normal glucose metabolism usually normalizes glycogen metabolism as
well.

In hypoglycemia caused by excessive insulin, liver glycogen levels are
high, but the high insulin level prevents the glycogenolysis necessary
to maintain normal blood sugar levels. Glucagon is a common treatment
for this type of hypoglycemia.
http://en.wikipedia.org/wiki/Glycogen"

Hunger is a feeling experienced when the glycogen level of the liver
falls below a threshold, usually followed by a desire to eat. The
usually unpleasant feeling originates in the hypothalamus and is
released through receptors in the liver.
http://en.wikipedia.org/wiki/Hunger"

If this were true, folks who are low-carbing would be hungrier but
they are not.

Is it correct and healthful to manipulate sysnthesis or energy stores
and resist/restrict their breakdown by added insulin in diabetics2?

The reason for administering insulin to a type-2 diabetic is simply to
lower blood glucose.

Which energy store need oxidation process?

Without oxygen for oxidation, you would die.
Will it promote free radical, inflammatory activities?
Andrew
--
Andrew B. Chung, MD/PhDhttp://EmoryCardiology.com- Hide quoted text -

- Show quoted text -

convicted neighbor Kumar wrote:
Andrew, in the Holy Spirit, boldly wrote:
convicted neighbor Kumar wrote:
Andrew, in the Holy Spirit, boldly wrote:
convicted neighbor kumar wrote:
Andrew, in the Holy Spirit, boldly wrote:
convicted neighbor Kumar wrote:

"Disorders of glycogen metabolism
The most common disease in which glycogen metabolism becomes abnormal
is diabetes, in which, because of abnormal amounts of insulin, liver
glycogen can be abnormally accumulated or depleted. Restoration of
normal glucose metabolism usually normalizes glycogen metabolism as
well.

In hypoglycemia caused by excessive insulin, liver glycogen levels are
high, but the high insulin level prevents the glycogenolysis necessary
to maintain normal blood sugar levels. Glucagon is a common treatment
for this type of hypoglycemia.
http://en.wikipedia.org/wiki/Glycogen"

Hunger is a feeling experienced when the glycogen level of the liver
falls below a threshold, usually followed by a desire to eat. The
usually unpleasant feeling originates in the hypothalamus and is
released through receptors in the liver.
http://en.wikipedia.org/wiki/Hunger"

If this were true, folks who are low-carbing would be hungrier but
they are not.

Is it correct and healthful to manipulate sysnthesis or energy stores
and resist/restrict their breakdown by added insulin in diabetics2?

The reason for administering insulin to a type-2 diabetic is simply to
lower blood glucose.

Which energy store need oxidation process?

Without oxygen for oxidation, you would die.

Will it promote free radical, inflammatory activities?

Reactive oxygen species are less likely when there are more reducing
equivalents around such as NADH and NADPH which would be with a diet
that has a greater proportion of carbohydrates.

Andrew
--
Andrew B. Chung, MD/PhD
http://EmoryCardiology.com

Andrew B. Chung, MD/PhD wrote:
convicted neighbor Kumar wrote:
Andrew, in the Holy Spirit, boldly wrote:
convicted neighbor Kumar wrote:
Andrew, in the Holy Spirit, boldly wrote:
convicted neighbor kumar wrote:
Andrew, in the Holy Spirit, boldly wrote:
convicted neighbor Kumar wrote:

"Disorders of glycogen metabolism
The most common disease in which glycogen metabolism becomes abnormal
is diabetes, in which, because of abnormal amounts of insulin, liver
glycogen can be abnormally accumulated or depleted. Restoration of
normal glucose metabolism usually normalizes glycogen metabolism as
well.

In hypoglycemia caused by excessive insulin, liver glycogen levels are
high, but the high insulin level prevents the glycogenolysis necessary
to maintain normal blood sugar levels. Glucagon is a common treatment
for this type of hypoglycemia.
http://en.wikipedia.org/wiki/Glycogen"

Hunger is a feeling experienced when the glycogen level of the liver
falls below a threshold, usually followed by a desire to eat. The
usually unpleasant feeling originates in the hypothalamus and is
released through receptors in the liver.
http://en.wikipedia.org/wiki/Hunger"

If this were true, folks who are low-carbing would be hungrier but
they are not.

Is it correct and healthful to manipulate sysnthesis or energy stores
and resist/restrict their breakdown by added insulin in diabetics2?

The reason for administering insulin to a type-2 diabetic is simply to
lower blood glucose.

Which energy store need oxidation process?

Without oxygen for oxidation, you would die.

Will it promote free radical, inflammatory activities?

Reactive oxygen species are less likely when there are more reducing
equivalents around such as NADH and NADPH which would be with a diet
that has a greater proportion of carbohydrates.
Means, Reactive oxygen species will be more, when energy **stores are
need to be broken?

Whether not using of energy stores and just fullfilling energy
requirements by glucose is helthful or unhealthful?

Is it normal and better to metabolize energy stores routinely or not?
Does it cause some abnormality of not using energy stores for
prolonged time by just depending on glucose in blood?
Andrew
--
Andrew B. Chung, MD/PhD
http://EmoryCardiology.com

convicted neighbor Kumar wrote:
Andrew, in the Holy Spirit, boldly wrote:
convicted neighbor Kumar wrote:
Andrew, in the Holy Spirit, boldly wrote:
convicted neighbor Kumar wrote:
Andrew, in the Holy Spirit, boldly wrote:
convicted neighbor kumar wrote:
Andrew, in the Holy Spirit, boldly wrote:
convicted neighbor Kumar wrote:

"Disorders of glycogen metabolism
The most common disease in which glycogen metabolism becomes abnormal
is diabetes, in which, because of abnormal amounts of insulin, liver
glycogen can be abnormally accumulated or depleted. Restoration of
normal glucose metabolism usually normalizes glycogen metabolism as
well.

In hypoglycemia caused by excessive insulin, liver glycogen levels are
high, but the high insulin level prevents the glycogenolysis necessary
to maintain normal blood sugar levels. Glucagon is a common treatment
for this type of hypoglycemia.
http://en.wikipedia.org/wiki/Glycogen"

Hunger is a feeling experienced when the glycogen level of the liver
falls below a threshold, usually followed by a desire to eat. The
usually unpleasant feeling originates in the hypothalamus and is
released through receptors in the liver.
http://en.wikipedia.org/wiki/Hunger"

If this were true, folks who are low-carbing would be hungrier but
they are not.

Is it correct and healthful to manipulate sysnthesis or energy stores
and resist/restrict their breakdown by added insulin in diabetics2?

The reason for administering insulin to a type-2 diabetic is simply to
lower blood glucose.

Which energy store need oxidation process?

Without oxygen for oxidation, you would die.

Will it promote free radical, inflammatory activities?

Reactive oxygen species are less likely when there are more reducing
equivalents around such as NADH and NADPH which would be with a diet
that has a greater proportion of carbohydrates.

Means, Reactive oxygen species will be more, when energy **stores are
need to be broken?

Reactive oxygen species will be more prevalent when endogeneous anti-
oxidant levels are low.

Whether not using of energy stores and just fullfilling energy
requirements by glucose is helthful or unhealthful?

Our bodies are optimally designed for utilizing glucose as the primary
chemical energy source.

Is it normal and better to metabolize energy stores routinely or not?

It is better not to have energy stores in the form of visceral adipose
tissue (VAT).

Does it cause some abnormality of not using energy stores for
prolonged time by just depending on glucose in blood?

Energy stores in the form of VAT would continue to cause harm as long
as it is present.

Andrew
--
Andrew B. Chung, MD/PhD
http://EmoryCardiology.com

On Mar 1, 5:32 am, "Andrew B. Chung, MD/PhD"
wrote:
convicted neighbor Kumar wrote:
Andrew, in the Holy Spirit, boldly wrote:
convicted neighbor Kumar wrote:
Andrew, in the Holy Spirit, boldly wrote:
convicted neighbor Kumar wrote:
Andrew, in the Holy Spirit, boldly wrote:
convicted neighbor kumar wrote:
Andrew, in the Holy Spirit, boldly wrote:
convicted neighbor Kumar wrote:

"Disorders of glycogen metabolism
The most common disease in which glycogen metabolism becomes abnormal
is diabetes, in which, because of abnormal amounts of insulin, liver
glycogen can be abnormally accumulated or depleted. Restoration of
normal glucose metabolism usually normalizes glycogen metabolism as
well.

In hypoglycemia caused by excessive insulin, liver glycogen levels are
high, but the high insulin level prevents the glycogenolysis necessary
to maintain normal blood sugar levels. Glucagon is a common treatment
for this type of hypoglycemia.
http://en.wikipedia.org/wiki/Glycogen"

Hunger is a feeling experienced when the glycogen level of the liver
falls below a threshold, usually followed by a desire to eat. The
usually unpleasant feeling originates in the hypothalamus and is
released through receptors in the liver.
http://en.wikipedia.org/wiki/Hunger"

If this were true, folks who are low-carbing would be hungrier but
they are not.

Is it correct and healthful to manipulate sysnthesis or energy stores
and resist/restrict their breakdown by added insulin in diabetics2?

The reason for administering insulin to a type-2 diabetic is simply to
lower blood glucose.

Which energy store need oxidation process?

Without oxygen for oxidation, you would die.

Will it promote free radical, inflammatory activities?

Reactive oxygen species are less likely when there are more reducing
equivalents around such as NADH and NADPH which would be with a diet
that has a greater proportion of carbohydrates.

Means, Reactive oxygen species will be more, when energy **stores are
need to be broken?

Reactive oxygen species will be more prevalent when endogeneous anti-
oxidant levels are low.
Will these(ROCs) be more on more VAT or on more lipid peroxidation?
Whether not using of energy stores and just fullfilling energy
requirements by glucose is helthful or unhealthful?

Our bodies are optimally designed for utilizing glucose as the primary
chemical energy source.

Is it normal and better to metabolize energy stores routinely or not?

It is better not to have energy stores in the form of visceral adipose
tissue (VAT).
Whether lipid peroxidation also happens of VAT?
Does it cause some abnormality of not using energy stores for
prolonged time by just depending on glucose in blood?

Energy stores in the form of VAT would continue to cause harm as long
as it is present.
Can/do they get oxidised as lipid peroxidation?
Andrew
--
Andrew B. Chung, MD/PhDhttp://EmoryCardiology.com- Hide quoted text -

- Show quoted text -